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A slim doctor has no reason to doubt current dietary guidelines. "If everyone did what I do, they'd get the same results," the doctor thinks. So when you explain that you've tried everything — eaten less, exercised more, counted every calorie — but still can't lose weight, the doctor knows what the problem is: You're not really following the advice.
It's unspoken. Subtle. But you feel it.
Here's what's interesting: When doctors themselves become overweight, many discover that the official guidelines don't work — not even for those who know the system inside and out. That's when some of them start looking for what's really going on.
What if everything you've been told is incomplete?
You Know This Feeling
For years you've struggled. Eaten less than your slim friends. Exercised. Weighed every bite. Maybe the weight dropped at first, but then it stopped. Or worse — it all came back, with extra.
And the whole time you've been hungry. Constantly hungry. With cravings for sweets that nearly drive you mad.
Hunger is a fundamental survival mechanism. Anyone can skip meals briefly — it's actually healthy. But nobody can live year after year with their body screaming for food. The body isn't built for that.
Hunger always wins.
Eventually you give up. Accept that this is just how it is. Maybe that's acceptance. Or maybe it's just exhaustion.
When Reality Doesn't Match the Theory
Think about this:
We ignore all the overweight people who have followed the advice for years. Who eat less, move more — and still gain weight. Hungry all the time.
You know what the unspoken message is, right? "You're not trying hard enough. You lack discipline."
At the same time, we ignore all the slim people who eat until they're full. Maybe even ice cream and cake. Without gaining weight. Without needing iron discipline.
Maybe you were like that once. Could eat whatever you wanted when you were young. But suddenly — often in your thirties or forties — it changed. Your body started reacting differently. Did you suddenly lose all your willpower?
Or for women: You entered menopause, and suddenly the weight stuck. Did you suddenly become less disciplined?
It's clearly a hormonal change. Yet the experts still insist it's just about eating less and burning more.
We know that some bodies work differently. Farmers breed pigs that gain weight easier. Researchers breed mice that get fat on food that doesn't affect normal mice. We know that hormones matter.
Yet we insist that for humans, the only explanation is lack of discipline.
Your Body Is Like a Hybrid Car
Here's the truth nobody's properly explained:
Your body can run on two completely different fuel sources — just like a hybrid car that switches between electricity and gasoline.
One fuel is sugar (which all carbohydrates turn into after you eat them). The other is fat (both from your food and from your body's fat stores).
Here's the key: Your body can only store about one day's worth of sugar. Think of it like a small battery that runs out quickly. But your fat storage? That's like an enormous fuel tank — basically unlimited capacity.
And here's where it gets interesting: The switch between these two fuels is controlled by a hormone called insulin.
When insulin is high, it's like the switch is locked: "Use sugar only! Don't touch the fat!"
Why? Because too much sugar in your blood is dangerous. High blood sugar can damage blood vessels, nerves, your eyes, everything. So your body treats excess sugar as a priority threat.
When there's sugar in your bloodstream, insulin goes up to push it into your cells quickly. And that same insulin blocks fat-burning completely.
This system works perfectly when you occasionally eat carbohydrates. The problem is that your body can only store about one day's worth of sugar, so it can't just let sugar accumulate while burning fat. That excess sugar would damage your body.
Makes sense, right? Deal with the limited-storage, potentially dangerous fuel first.
What Went Wrong: Modern Food Broke the Switch
For most of human history, we never ate anywhere near the amount of carbohydrates we eat today. Natural fruits weren't nearly as sweet as today's cultivated ones. Before agriculture, we might go days between eating significant amounts of carbs.
Our bodies were built to switch easily between burning sugar (when available) and burning fat (the rest of the time). Scientists call this Metabolic Flexibility — a normal, healthy function.
But now? We bombard our bodies with Carbohydrates all day long. Bread for breakfast. Sandwich for lunch. Pasta for dinner. Snacks in between. Often it's refined carbs — white bread, sugar, juice — that get converted to sugar incredibly fast.
Your body responds the only way it can: By producing more and more insulin to handle all that sugar.
And remember — high insulin means the fat-burning switch is locked in the "off" position.
The Fuel Switch Gets Stuck
When you keep eating carbohydrates meal after meal, day after day, something starts to happen: Your cells become less responsive to insulin.
It's similar to what happens when you hear a loud sound continuously — eventually you tune it out. Your cells, constantly exposed to high insulin, start ignoring the signal. Scientists call this Insulin Resistance.
But here's the problem: High Blood Sugar is dangerous, so your body can't just accept defeat. Instead, it shouts louder — producing even more insulin to force the sugar into your cells.
Some people's bodies can produce 200-300% more insulin than normal. Some even reach 900% over normal levels, trying desperately to overcome the resistance.
And all that extra insulin? It keeps the fuel switch locked. You physically cannot burn fat as long as insulin is high.
Think about it: You're walking around with a massive energy reserve — all that stored fat. But it's like having a full gas tank in that hybrid car while the switch is stuck on "battery only." The energy is right there, but you can't access it.
The Cruel Paradox
Your muscles need energy. Your brain needs energy. But with the fuel switch stuck, they can't access the fat stores. And because of insulin resistance, your cells struggle to use sugar efficiently — even though there's plenty of it.
What happens then? Your body activates every survival mechanism it has:
The hunger signals break down. Insulin interferes with Leptin, the hormone that says "I'm full, stop eating." When leptin can't do its job, you never feel truly satisfied.
At the same time, Ghrelin — the hunger hormone — goes up. Your body thinks it's starving (because it can't access the fat), so it screams at you to eat more.
But here's the really maddening part: Your body is in sugar-burning mode because of all the insulin, so it desperately craves sugar. That irresistible urge for something sweet? That's not weakness — that's your body screaming "I need fuel NOW!"
And your survival systems kick in further: Energy shortage detected? Slow down the metabolism — researchers have documented reductions up to 20-30%.
So you end up both hungry and tired, with intense sugar cravings, while plenty of energy sits locked away as fat.
This isn't a willpower problem. This is a hormone system overwhelmed by constant carbohydrate intake.
Why "Eat Less, Move More" Doesn't Work
Think about someone who gains just one kilo (2.2 pounds) per year from age 30 to 50. That's 20 kilos over twenty years. Sound familiar?
According to "calories in, calories out" logic, that means they ate too much. But how much too much?
One kilo of fat contains about 9000 calories. Spread over 365 days, that's about 24 calories per day more than they burned.
That's roughly three almonds. Or half a teaspoon of olive oil.
If the problem really is just math — calories eaten minus calories burned — then why can't you just eat three almonds less each day? Why aren't we all either rail-thin or enormous?
Because the real problem isn't the arithmetic. It's the fuel switch.
When your switch is working properly, your body naturally regulates appetite and energy to maintain balance. You eat when you're hungry, stop when you're full, and your weight stays stable.
You don't need willpower. Your slim friend isn't constantly resisting temptation — they just feel full and naturally stop eating. They don't have the same cravings as you.
But when the switch is stuck — when insulin resistance has set in — the same amount of food that keeps your slim friend energized makes you store fat and feel hungry at the same time. Because the energy goes straight into fat deposits instead of to your muscles and organs that need it.
It's not three almonds that make the difference. It's whether your body can access its fuel and whether you feel hungry or satisfied.
The Invisible Problem Your Doctor Doesn't Test For
Here's the scary part: You can have completely normal blood sugar and still have dangerously high insulin.
Your doctor checks your blood sugar. "Looks fine!" But nobody measures your insulin.
You can live with what researchers call Metabolic Syndrome — essentially a stuck fuel switch — for 20-30 years before it turns into Prediabetes or Type 2 Diabetes
Why the difference? Because there's huge variation in how much insulin different bodies can produce.
If you're "good" at producing massive amounts of insulin (400-900% over normal), you can keep blood sugar normal even with severe insulin resistance. You gain weight fast, feel hungry all the time — but your blood sugar stays normal. The doctor sees no problem.
Here's the really sad part: The same doctors who insist you just need to eat less know perfectly well that when they give type 2 diabetics more insulin to lower blood sugar, those patients typically gain even more weight.
They also know that people with type 1 diabetes (who produce no insulin at all) lose massive amounts of weight before diagnosis — no matter how much they eat. And when they finally get insulin, they start gaining weight again.
Doctors know insulin drives fat storage. Yet they still call weight gain a discipline problem.
So There's Actually Hope?
Yes. And here's why:
When you understand that the real problem is a stuck fuel switch caused by chronically high insulin, the solution becomes clear:
You need to let insulin drop so the switch can unlock.
How? By drastically reducing the thing that raises insulin: Carbohydrates.
Not a little. A lot. Down to around 20-50 grams per day. (For context: one slice of bread has about 15 grams, a medium banana around 27 grams, a cup of rice about 45 grams.)
The exact amount is individual. The magic happens when insulin drops low enough that your body switches to fat burning. When this happens, your body produces Ketones — measurable compounds that signal fat burning is happening.
If you're very insulin resistant, you might need to go lower with carbs at first. If you're less resistant — and especially if you exercise regularly — you can handle slightly more.
Be Patient with Yourself
Results might not be instant. You may need to adjust and find your personal threshold.
And here's something many don't talk about: Changing habits is hard. And some people have developed a genuine carbohydrate addiction.
Sugar stimulates the same brain centers as certain narcotics. If you've been eating a lot of sugar and refined carbs for years, your brain has adapted to expect that dopamine hit.
For some people, gradually reducing sugar doesn't work well — you have to go "cold turkey." The first few weeks can feel like withdrawal. You might experience headaches, irritability, intense cravings. This is real.
But here's the good news: It passes. Usually within 2-3 weeks, those cravings disappear. Completely.
When you do this, something happens:
Insulin levels plummet. Often within days.
The fuel switch unlocks. Your body can finally access the fat it's been storing.
The hunger disappears. Not gradually. Often within the first week or two. Not because you're training willpower, but because Leptin and Ghrelin can finally function normally again. And those irresistible sugar cravings? Gone.
Energy returns. Because your body now has access to fuel from your fat stores.
Research shows insulin sensitivity can improve by up to 75% in just 14 days. Many see marked improvements in metabolic markers within 90 days.
And here's something fascinating: Studies of overweight people with insulin resistance showed that when they switched to very low-carb diets — and were allowed to eat as much as they wanted — they automatically reduced their intake to around 2000 calories per day. And felt more satisfied than before (when they ate over 3000 calories per day).
Not less satisfied. More satisfied.
After the initial adjustment period, it's not willpower. It's biochemistry.
What Happens Next
This is where the practical details come in — what to eat, how to start, what to expect. But that's too much for this article.
The important thing right now is understanding why this works. Understanding that:
- It's not about willpower or discipline — it's about biology and hormones
- Hunger always wins — and that's normal, not a character flaw
- You're not lazy or greedy — you're fighting a fuel switch stuck by constant intake of Carbohydrates
- There's a way out — not through heroic self-control, but by working with your body's chemistry
In our next article, we'll cover exactly how to get started: what foods to eat, what to avoid, how to know if it's working, and what to watch out for (especially if you're on medication).
But for now, just know this:
The struggle was never about willpower. Your body wasn't designed to handle the amount of carbohydrates that modern food throws at it. Once that fuel switch gets stuck, even the most disciplined person will struggle with constant hunger.
But when you understand what's really happening, you can fix it.
Your next meal could be the start of something different.
Next Steps
Books
- Gary Taubes. (2020). The Case for Keto: The Truth About Low-Carb, High-Fat Eating. Granta Books. ISBN: 178378654X 9781783786541.
Research Papers
- G Boden, K Sargrad, C Homko, M Mozzoli, T P Stein. (2005). "Effect of a low-carbohydrate diet on appetite, blood glucose levels, and insulin resistance in obese patients with type 2 diabetes". Annals of internal medicine, 142(6). https://doi.org/10.7326/0003-4819-142-6-200503150-00006.
- C B Ebbeling, H A Feldman, G L Klein, J M Wong, L Bielak, S K Steltz, P K Luoto, R R Wolfe, W W Wong, D S Ludwig. (2018). "Effects of a low carbohydrate diet on energy expenditure during weight loss maintenance: randomized trial". BMJ (Clinical research ed.), 363. https://doi.org/10.1136/bmj.k4583.
- Alison B. Evert, Michelle Dennison, Christopher D. Gardner, W. Timothy Garvey, Ka Hei Karen Lau, Janice MacLeod, Joanna Mitri, Raquel F. Pereira, Kelly Rawlings, Shamera Robinson, Laura Saslow, Sacha Uelmen, Patricia B. Urbanski, William S., Jr. Yancy. (2019). "Nutrition Therapy for Adults With Diabetes or Prediabetes: A Consensus Report". Diabetes Care, 42(5), 731-754. https://doi.org/10.2337/dci19-0014.
- Richard D. Feinman, Wendy K. Pogozelski, Arne Astrup, Richard K. Bernstein, Eugene J. Fine, Eric C. Westman, Anthony Accurso, Lynda Frassetto, Barbara A. Gower, Samy I. McFarlane, Jörgen Vesti Nielsen, Thure Krarup, Laura Saslow, Karl S. Roth, Mary C. Vernon, Jeff S. Volek, Gilbert B. Wilshire, Annika Dahlqvist, Ralf Sundberg, Ann Childers, Katharine Morrison, Anssi H. Manninen, Hussain M. Dashti, Richard J. Wood, Jay Wortman, Nicolai Worm. (2015). "Dietary carbohydrate restriction as the first approach in diabetes management: Critical review and evidence base". Nutrition, 31(1), 1-13. https://doi.org/10.1016/j.nut.2014.06.011.
- Kelley DE, Mandarino LJ. Fuel selection in human skeletal muscle in insulin resistance: a reexamination. Diabetes. 2000;49(5):677-83. https://doi.org/10.2337/diabetes.49.5.677
- Smith RL, Soeters MR, Wüst RCI, Houtkooper RH. Metabolic Flexibility as an Adaptation to Energy Resources and Requirements in Health and Disease. Endocr Rev. 2018;39(4):489-517. https://doi.org/10.1210/er.2017-00211
- Goodpaster BH, Sparks LM. Metabolic Flexibility in Health and Disease. Cell Metab. 2017;25(5):1027-1036. https://doi.org/10.1016/j.cmet.2017.04.015
- Klok MD, Jakobsdottir S, Drent ML. The role of leptin and ghrelin in the regulation of food intake and body weight in humans: a review. Obes Rev. 2007;8(1):21-34. https://doi.org/10.1111/j.1467-789X.2006.00270.x